Download Free Medical GLAUCOMA PowerPoint Presentation

GLAUCOMA PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Regardless of the intraocular pressure, it is defined by characteristic optic neuropathy consistent with Excavation and undermining of the neural and connective tissue elements of the optic disc and eventual development of characteristic visual field defects. Learn about the three factors that determine Intraocular pressure.

Tags : GLAUCOMA, intraocular pressure,Canal of Schlemm, episcleral venous pressure, aqueous humor production, Uvoscleral drainage, Iris

Published on : Dec 10, 2020 | Views : 13158 | Downloads : 178

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

GLAUCOMA Kyren Janne M. Ladao
Slide 2-

Objectives: To define glaucoma 1 To differentiate the classifications of glaucoma 2 3 To diagnose glaucoma 4 To discuss the different treatment for glaucoma
Slide 3-

Glaucoma Definition
Slide 4-

Glaucoma Regardless of the intraocular pressure, it is defined by characteristic optic neuropathy consistent with: Excavation and undermining of the neural and connective tissue elements of optic disc Eventual development of characteristic visual field defects
Slide 5-

Three factors that determine the Intraocular pressure Rate of aqueous humor production Resistance of aqueous outflow across trabecular meshwork – Canal of Schlemm Level of episcleral venous pressure
Slide 6-

Three factors that determine the Intraocular pressure Rate of aqueous humor production Resistance of aqueous outflow across trabecular meshwork – Canal of Schlemm Level of episcleral venous pressure
Slide 7-

Aqueous humor flow
Slide 8-

Trabecular outflow Uvoscleral drainage Iris
Slide 9-

In Glaucoma
Slide 10-

Glaucoma Classification
Slide 11-

Not associated with known ocular or systemic disorder that cause increased resistance to aqueous outflow or angle closure Usually bilateral Primary Glaucoma Associated with known ocular or systemic disorder responsible for decreased aqueous outflow Usually unilateral Secondary Glaucoma
Slide 12-

Insert the title of your subtitle Here Most common Angle is wide open Slow blockage of drainage canal → GRADUAL increase in IOP → atrophy of outer rim of optic nerve → decrease in peripheral vision → central vision loss Open Angle Glucoma Angle is narrow Lens being pushed up against iris → drainage gets blocked → SUDDEN increase in IOP → severe eye pain, redness, BOV, visual halos → vision loss Close Angle Glucoma
Slide 13-

Primary Angle Closure Suspect (PAS) Angle closure Angle closure Increased IOP Primary Angle Closure (PAC) Primary Angle Closure Progressive (PACP) Angle closure Increased IOP Optic neuropathy Closed Angle Glaucoma
Slide 14-

Glaucoma Diagnosis
Slide 15-

1 2 3 4 Measurement of pressure in the eyes Tonometry Used to visualize the anterior chamber angle Gonioscopy Using perimetry to clinical assess the visual field Pathological cupping is caused by an irreversible decrease in the number of nerve fibres, glial cells and blood vessels. Optic nerve inspection Visual field testing
Slide 16-

1 2 4 Measurement of pressure in the eyes Tonometry Using perimetry to clinical assess the visual field Pathological cupping is caused by an irreversible decrease in the number of nerve fibres, glial cells and blood vessels. Optic nerve inspection Visual field testing
Slide 17-

1 2 3 Measurement of pressure in the eyes Tonometry Used to visualize the anterior chamber angle Gonioscopy Using perimetry to clinical assess the visual field Visual field testing
Slide 18-

ppt slide no 18 content not found
Slide 19-

2 3 4 Used to visualize the anterior chamber angle Gonioscopy Using perimetry to clinical assess the visual field Pathological cupping is caused by an irreversible decrease in the number of nerve fibres, glial cells and blood vessels. Optic nerve inspection Visual field testing
Slide 20-

1 3 4 Measurement of pressure in the eyes Tonometry Used to visualize the anterior chamber angle Gonioscopy Pathological cupping is caused by an irreversible decrease in the number of nerve fibres, glial cells and blood vessels. Optic nerve inspection
Slide 21-

Glaucoma Treatment
Slide 22-

Medications ẞ- adrenergic receptor antagonist Carbonic anhydrase inhibitors Decrease production of aqueous humor Prostaglandin analogs Increase outflow of aqueous humor A-adrenergic agonist Decrease production and increase outflow
Slide 23-

Medications ẞ- adrenergic receptor antagonist Carbonic anhydrase inhibitors Decrease production of aqueous humor Prostaglandin analogs Increase outflow of aqueous humor A-adrenergic agonist Decrease production and increase outflow ẞ- adrenergic receptor antagonist Timolol Betaxolol
Slide 24-

Medications ẞ- adrenergic receptor antagonist Carbonic anhydrase inhibitors Decrease production of aqueous humor Prostaglandin analogs Increase outflow of aqueous humor A-adrenergic agonist Decrease production and increase outflow Carbonic anhydrase inhibitors Dorzolamide Acetazolamide
Slide 25-

Medications ẞ- adrenergic receptor antagonist Carbonic anhydrase inhibitors Decrease production of aqueous humor Prostaglandin analogs Increase outflow of aqueous humor A-adrenergic agonist Decrease production and increase outflow Prostaglandin analogs Latanoprost Bimatoprost
Slide 26-

Medications ẞ- adrenergic receptor antagonist Carbonic anhydrase inhibitors Decrease production of aqueous humor Prostaglandin analogs Increase outflow of aqueous humor A-adrenergic agonist Decrease production and increase outflow A-adrenergic agonist Brimonidine Apraclonidine
Slide 27-

Laser Treatment Trabeculoplasty Name Here Programmer Iridotomy Iridotomy
Slide 28-

Thank you!!!