Download Free Medical Intestinal Obstruction PowerPoint Presentation

Intestinal Obstruction PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical presentation on Digestive System, which is titled "Intestinal Obstruction", to know about intestinal obstruction, one of the common cause of acute abdomen.

Tags : Digestive System, Intestinal Obstruction, acute abdomen

Published on : Jul 10, 2021 | Views : 4036 | Downloads : 31

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Dr. Pankaj Kumar Assistant Professor Surgical Gastroenterology
Slide 2-

One of the common cause of acute abdomen May lead to high morbidity and mortality if not treated correctly It can be classified into two types: Dynamic (mechanical) Adynamic
Slide 3-

Dynamic: where peristalsis is working against a mechanical obstruction. Adynamic: mechanical element is absent - Peristalsis my be absent(paralytic ileus) -May be present in non propulsive form. (mesenteric vascular occlusion or pseudo-obstruction)
Slide 4-

DYNAMIC 1.Intraluminal: impacted faeces, foreign bodies, gallstones, Bezoars. 2.Intramural: tumors, inflammatory strictures, 3.Extramural: adhesion, hernias, volvulus, intussusception, tumors
Slide 5-

also can be divided into: 1. Small bowel obstruction (SBO) -high ->early perfuse vomiting rapid dehydration -low->predominant pain, and central distention Vomiting delayed multiple central air-fluid levels seen on AXR 2. Large bowel obstruction (LBO) early pronounced distension, mild pain vomiting, dehydration late e.g. -carcinoma -diverticulitis or volvulus
Slide 6-

Acute obstruction:- usually in small bowel -obstruction with severe colicky central abdominal pain, distension, early vomitting and constipation. Chronic obstruction: -usually in large bowel - lower abdominal colic & obstipation followed by distension. Acute on chronic: short history of distension & vomiting against background of pain & constipation. Subacute obstruction : incomplete obstruction.
Slide 7-

Simple: blockage without interfering with vascular supply Strangulation: significant impairment of blood supply most commonly associated with hernia, volvulus, intussusception, mesentric infarction, adhesions/Bands -surgical emergency Closed loop obstruction: bowel is obstructed at both the proximal and distal end.
Slide 8-

Adhesions- 40% Tumors -15% Inflamatory- 15% Obstructed hernia-12% Intraluminal-10% Miscellaneous -8%
Slide 9-

Irrespective of etiology or acuteness of onset: Proximal to obstruction Increased fluid secretion  abdominal distention Accumulation of gas  abdominal distention Increased intraluminal pressure Vomiting Dehydration Dilatation of bowel Reflex contraction of smooth muscle  colicky pain Increased peristalsis to overcome obstruction  increased bowel sounds If obstruction not overcome  bowel atony Decreased reabsorption with time and flaccidity to prevent vascular damage from high pressure Distal to obstruction: nothing is passed & bowel collapse  constipation
Slide 10-

The four cardinal features of intestinal obstruction: -abdominal pain -vomiting -distension -constipation Vary according to:- location of obstruction Duration of obstruction underlying pathology intestinal ischemia
Slide 11-

Abdominal pain - colicky in nature, around the umbilicus in SBO while in the lower abdomen in LBO - if it becomes continuous, think about perforation or strangulation. - does not usually occurs in paralytic ileus. Vomiting -starts early in SBO and late in LBO -As obstruction progresses vomitus alters from digested food to faeculent due to enteric bacterial overgrowth Distension -more with lower obstruction
Slide 12-

Constipation -more with lower or complete obstruction - constipation is either absolute (no feces or flatus) cardinal feature of complete Int.Obst. or relative (flatus passed). it does not apply in -Richter’s Hernia -Gallstone obturation. -mesentric vascular occlusion. - obstruction associated with pelvic abscess. -diarrhea may be present with partial obstruction
Slide 13-

Dehydration More common in small bowel obstruction.due to repeated vomiting . Secondary polycythemia due to raised B.urea & hematocrit. Pyrexia Onset of ischemia. Intestinal perforation. Inflamation associated with int. obst.
Slide 14-

In strangulation: severe constant abdominal pain fever tachycardia tenderness with rigidity/rebound tenderness. shock
Slide 15-

General examination- Vital signs Signs of dehydration –tachycardia, hypotension dry mucus membrane, decreased skin turgor, decreased urine output Inspection distension, scars, peristalsis, masses, hernial orifices Palpation tenderness, masses, rigidity Percussion tympanitic abdomen Auscultation high pitched bowel sound or silent abdomen *Examine rectum for mass, blood, feces or it may be empty in case of complete obstruction
Slide 16-

Hemogram - WBC (neutrophilia-strangulation) Hyper kalemia, hyperamylasemia & raised LDH may be associated with stangulation. Plain AXR Sigmoidoscopy (carcinoma, volvulus) Contrast x-ray CT abdomen.
Slide 17-

When distended by gas: Jejunum is characterized by valvulae conniventes(completely pass across the width & regularly placed) Ileum is featureless. Caecum is shown by rounded gas shadow in RIF. Colon shows haustral folds. Fluid level appears later than gas shadow Two fluid level in small bowel considered normal. No. of fluid level is proportional to degree of obstruction and distal site in small bowel.
Slide 18-

Colonic obstruction does not commonly give rise to small bowel fluid level unless advanced. Associated with large ammount of gas in caecum. Ba-follow through is contraindicated in acute intestinal obstruction.
Slide 19-

Three main measures- - GI drainage Fluid &Electrolyte replacement - Relief of obstruction, usually surgical
Slide 20-

Treatment Conservative: -Nasogastric aspiration by Ryles tube -IV fluids- volume varies depending on dehydration -NPO -urinary catheter -check temp. and pulse 2 hourly -abdominal examination 8 hourly -Broad spectrum antibiotics initiated early- reduce bacterial overgrowth.
Slide 21-

Some cases will settle by using this conservative regimen, other need surgical intervention. Surgery should be delayed till resuscitation is complete unless signs of strangulation and evidence of closed-loop obstruction. Cases that show reasons for delay should be monitored continuously for 72 hours in hope of spontaneous resolution e.g. adhesions with radiological findings but no pain or tenderness “The sun should not both rise and set” in cases of unrelieved obstruction.
Slide 22-

Indication for surgery: - failure of conservative management - tender, irreducible hernia - strangulation If the site of obstruction is unknown; laparotomy assessment is directed to- -The site of obstruction. -The nature of obstruction. -The viability of gut. The site of obstruction can be determined by caecum
Slide 23-

Surgical treatment Operative decompression required-if dilatation of bowel loops prevent exposure, bowel wall viability is compromised, or if subsequent closure will be compromised. Savage’s decompressor used within seromuscular purse-string suture. Or large-bore NG tube maybe used for milking intestinal contents into stomach.
Slide 24-

The type of surgical procedure depend upon the cause of obstruction viz division of bands,adhesiolysis, excision ,or bypass *Once obstruction relieved, the bowel is inspected for viability, and if non-viable, resection is required. Indication of non-viability 1.absent peristalsis 2.loss of normal shine 3.loss of pulsation in mesentry 4.green or black color of bowel
Slide 25-

If in doubt of viability, bowel is wrapped in hot packs for 10 minutes with increased oxygen and reassessed for viability. Resection of non viable gut should be done followed by stoma. Sometimes a second look laprotomy is required in 24-48 hours e.g. multiple ischemic areas.
Slide 26-

Most common cause of intestinal obstruction. Peritoneal irritation results in local fibrin production, which produce adhesions. BANDS Congenital : obliterated vitellointestinal duct. A string band following previous bacterial peritonitis. A portion of greater omentum adherent to parietes.
Slide 27-

Causes of adhesions : Abdominal operation : anastomosis, raw peritoneal surfaces Foreign material: talc, starch, gauze, silk Infection: peritonitis, T.B. Inflammatory conditions: crohn’s disease. Radiation entritis. Prevention Good surgical technique. Washing the peritoneal cavity with saline to remove the clots. Minimizing contact with gauze. Covering the anastomosis & raw peritoneal surfaces.
Slide 28-

Usually conservative treatment is curative. (i.v. rehydration & nasogastric decompression) It should not be prolonged beyond 72 hrs. Surgery Division of band. Minimal adhisiolysis.
Slide 29-

Repeat adhesiolysis alone. Noble’s plication : adjacent intestinal coils (15-20 cms) are sutured with serosal sutures. Charles-Phillips trans-mesenetric plication. Intestinal intubation : initraluminal tube insertion via a WITZEL jejunostomy or gastrostomy.
Slide 30-

When a portion of small intestine is entrapped in one of retropritoneal fossae or in a congenital mesentric defect. Sites of internal herniation: Foramen of winslow. A hole in mesentry / transverse mesocolon. Defects in broad ligaments. Congenital/ acquired diaphragmatic hernia. Duodenal retroperitoneal fossae- Lt. paraduodenal & rt. Duodenoojejunal. intersigmoid fossae.
Slide 31-

It is uncommon in the absence of adhesions. Treatment : to release the constricting agent by division.
Slide 32-

It tends to occur in elderly. Erosion of large gallstone into duodenum. Present with recurrent obstruction. X-ray: small bowel obstruction with air in billiary tree. -may show a radio opaque gall stone. Treatment : laparotomy & removal /crushing of stone.
Slide 33-

After partial /total gastrectomy. Unchewed food can cause obstruction. Treatment similar to gall stone. BEZOARS Trichobezoars Phytobezoars WORMS Ascaris lumbricoides Frequently follows initiation of antihelminthic therapy. Eosinophilia/worm with in gas filled bowel loops. Laparotomy.
Slide 34-

One portion of gut becomes invaginated with in adjacent segment. Most common in children(3-9 months.) Ideopathic-70% Associated gastroenteritis/UTI- 30% Hyperlpasia of Peyer’s patches in terminal ileum can be initiating factor.
Slide 35-

In older children intussusception is usually associated with a lead point – meckel’s diverticulum, polyp, & appendix. Adults: always with a lead point.- polyp, submucosal lipoma/ tumor. It is composed of three parts: -Entering/ inner tube(Intussusceptum) - Returning/ middle tube -Sheath/ outer tube(intussuscipiens) It is an example of strangulating obstruction with impaired blood supply of inner layer. It may be ileoileal(5%); ileocolic(77%); ileo-ileo-colic(12%); colocolic (2%) & multiple.
Slide 36-

Severe colic pain. vomitting as time progress blood & mucus (the ‘redcurrent’ jelly stool). Abdominal lump(sausage shaped) Emptiness in RIF(the sign of Dance). Death may occur from bowel obstruction or peritonitis secondary to gangrene.
Slide 37-

Plain X-ray Abd.: Bowel obstruction with absent caecal shadow gas in ileo-ileal & ileo-colic cases. Ba-enema: the claw sign in ileocolic & colocolic cases. CT scan in equivocal cases of ileo-ileal intussusception. (small bowel mass may be revealed) Differential Diagnosis Acute enterocolitis: faecal matter/ bile is always present. Henoch-schoenlein purpura. Rectal prolapse: projecting mucosa can be felt in continuity with perianal skin
Slide 38-

Theraputic Ba-enema : -in infants. - unlikely to succeed in lead points. - contrindications: peritonism, prolonged history (> 48 hrs.). Operative After resuscitation ;Laparotmy with reduction. Cope’s method. Irreducible/ gangrenous intussusception: excision of mass & anastomosis.
Slide 39-

to be cont……