Download Free Medical Case of Perforated Diverticulitis PowerPoint Presentation

Case of Perforated Diverticulitis PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Case of Perforated Diverticulitis

Tags : Diverticulitis

Published on : Aug 08, 2021 | Views : 2373 | Downloads : 3

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Case of Perforated Diverticulitis Maedeh Dahaghin
Slide 2-

History A female patient named F.Ef aged 86 from Tehran and has 3 offsprings, housewife and has no spesial habits.
Slide 3-

Complaints Constant and non -positional abdominal pain Constipate Insomnia Anorexia Nausia and Vomiting
Slide 4-

History of the present illness The constipation started 4 days before admission abdominal pain that is constant and non-positional. the EKG led to AF and the reason of spO2=83% is Pulmonary edema. GCS 15/15 SpO2:83% PR:73% BP:100/70 Vital signs:
Slide 5-

Past history PMH Hemorrhoid Constipation angiography Other active problems: Pulmonary edema Unilateral renal atrophy AF PDH Nitroglycerin Atorvastatin ASA Hydrochlorothiazide Carvedilol
Slide 6-

Examination We need CT but We have a problem Due to GFR=25 , the patient is at high risk of receiving a contrast agent but is necessary so following check up is crucial: ✔Check of BUN, Na, k daily until 72h after CT ✔Use the least amount of contrast agent
Slide 7-

Diagnosis after CT scan Perforated Diverticulitis
Slide 8-

Diverticula • Small (0.5 - 1.0 cm) pouches protruding from bowel wall • Most pseudo diverticula: mucosa and submucosa only- muscle layer not present • True diverticula: all layers of the bowel wall involved • Up to 60% of people living in industrialized countries will develop colonic diverticula.
Slide 9-

Pathophysiology Diverticular Disease • Increased intraluminal pressure • Caused by low fiber, constipation • Sigmoid colon most commonly involved (95%) • Smallest diameter • Laplace’s law: generates highest pressure • Incidence of diverticular disease increases with age: • 30% at age 60 • 60-80% at age 80
Slide 10-

Risk factors Low fiber Diet Smoking Constipation Obesity Kidney Disease Coronary Heart Disease Hyperlipidemia Hypothyroidism Diabetes Mellitus
Slide 11-

Clinical Classification Uncomplicated vs. Complicated • Uncomplicated : Pericolic soft-tissue stranding colonic wall thickening phlegmon • Complicated: Acute diverticulitis + Abscess Obstruction Perforation Fistula
Slide 12-

ppt slide no 12 content not found
Slide 13-

Presentation • Symptoms • LLQ Pain, Fever, Diarrhea or constipation, Urinary symptoms if inflammation adjacent to the bladder •Classic Triad :Fever, Leukocytosis, LLQ tenderness Free perforation: peritonitis, sepsis
Slide 14-

Management Complicated Diverticulitis: Free perforation • 1% to 2% of cases • Mortality between 20% - 30% • Hinchey Stage III - Purulent peritonitis • Hinchey Stage IV - free perforation with fecal peritonitis
Slide 15-

According to current ASCRS guidelines, HP recommended Emergent Surgical Intervention for Management of Hinchey III & IV disease
Slide 16-

Surgical Intervention the patient was anesthetized with Etomidate and intubated. Injection of Norepinephrine to prevent hypotension, and NaHco3 to correct metabolic acidosis. PH:7.27, PCO2:39.2, HCO3:17 The patient came to ICU intubated with drip of norepinephrine.
Slide 17-

Patient Management (ICU) In the ICU Heparin was started in the early hours due to EKG changes and elevated troponin. Cardiac troponin is specific and sensitive biomarker of cardiac injury(MI). Heparin 500-600 u/h
Slide 18-

Troponin changes
Slide 19-

Management CO was between 0.5- 1 increase the preload. What can we do? increase the volume. hypotonic fluids such as saline half, dextrose 5% and 1/3 2/3 Isotonic solutions such as Ringer, lactate ringer and NaCl 0.9% or hypertonic solutions such as Albumin 20% and 5% sodium chloride are suitable.
Slide 20-

Sepsis, a recurring event occurs in hospitalized or who have recently been hospitalized. People in an intensive care unit sensitive to sepsis. Any infection, however, could lead to sepsis.
Slide 21-

Antibiotic therapy
Slide 22-

In this case: RR=15 SBP=95 GCS:3/15
Slide 23-

Septic shock With a mortality rate about 50% . Intervention should be performed in the first hour , otherwise organ damages will be increased.
Slide 24-

Sepsis management Sepsis management bundle to be implemented within 1 hour of onset of sepsis: Consider intravenous fluid resuscitation. Administer intravenous antibiotics. Consider intravenous fluid resuscitation. Use Norepinephrine and if CO Norepinephrine + epinephrine
Slide 25-

Vitamin C 1-Prevention of Sepsis Associated Encephalopathy ( SAE) 2-Endothelial stabilization 3-Reduce the required dose of vasopressors 4-Antioxidant effects
Slide 26-

Cardiogenic shock Due to the lost of myocardial tissue, the CO and the vascular resistance The extent of damage to the heart tissue is unpredictable .
Slide 27-

Treatment of cardiogenic shock Management : 1-inotropes: Insulin + Dextrose(D50W)+K or Milrinone Milrinone to patients with hypotension But inotropes are desirable, especially in this case It is a choise because PCWP is high and CO is low. This is an example of patients with chronic respiratory disease and heart failure who have high pulmonary arterial pressure. Also for those who have pulmonary edema Milrinone is a good medicine.
Slide 28-

Treatment of cardiogenic shock 2- Vasopressors :Norepinephrine, Epinephrine The patient could not tolerate cessation of norepinephrine for several days. 3-diuretic: Lasix The best diuretic in heart failure is spironolactone, and oral or injectable ferrosamide is the worst drug. We do not give it because activates the RAS system. We use injectable ferrosomide only in cases where the patient has congestion such as this case.
Slide 29-

A widely used combination in ICU K+ Insulin+Dextrose(D50W) It is a inotropic and combination of insulin, dextrose and potassium affects potassium channels and carries sugar into heart cells. And helps produce intracellular ATP
Slide 30-

Erythropoietin (Eprex®) 1-Activation of the inflammatory process in critical ill patient hepcidin Over expression 2-Inflammation Erythropoietin synthesis iron absorption
Slide 31-

Management of acidosis PH=7.25, PCO2=33.3, HCO3=14.9 First stage: eliminate the cause and establish ventilation. second stage: giving bicarbonate Some acidosis is allowed in the ICU
Slide 32-

Finally The patient expired…
Slide 33-

References 1- Cirocchi R, Arezzo A, Vettoretto N, et al. Role of damage control surgery in the treatment of Hinchey III and IV sigmoid diverticulitis: a tailored strategy. Medicine (Baltimore) 2014; 93:e184. 2- Tartaglia D, Costa G, Camillò A, et al. Damage control surgery for perforated diverticulitis with diffuse peritonitis: saves lives and reduces ostomy. World J Emerg Surg 2019; 14:19. 3-Sohn M, Iesalnieks I, Agha A, et al. Perforated Diverticulitis with Generalized Peritonitis: Low Stoma Rate Using a "Damage Control Strategy". World J Surg 2018; 42:3189. 4-Maggard MA, Zingmond D, O'Connell JB, Ko CY. What proportion of patients with an ostomy (for diverticulitis) get reversed? Am Surg 2004; 70:928. 5- Vermeulen J, Coene PP, Van Hout NM, et al. Restoration of bowel continuity after surgery for acute perforated diverticulitis: should Hartmann's procedure be considered a one-stage procedure? Colorectal Dis 2009; 11:619. 6- Banerjee S, Leather AJ, Rennie JA, et al. Feasibility and morbidity of reversal of Hartmann's. Colorectal Dis 2005; 7:454