Download Free Medical Surgical Infections PowerPoint Presentation

Surgical Infections PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical presentation on Surgical Site Infections, which is titled "Surgical Infections", to know about surgical infections.

Tags : Surgical Site Infections, Surgical Infections

Published on : Jul 05, 2021 | Views : 4292 | Downloads : 21

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Surgical Infections Awadh Alqahtani MD,MSc,FRCSC(surgery)FRCSC(oncology) FISC Surgical oncologist and laparoscopic Bariatric surgeon
Slide 2-

Infection Infection is defined by: Microorganisms in host tissue or the bloodstream Inflammatory response to their presence.
Slide 3-

ppt slide no 3 content not found
Slide 4-

Inflammatory Response Localized: Rubor, Calor, Dolor, Tumor, and functio laesa (loss of function) Systemic: Systemic Inflammatory Response Syndrome (SIRS)
Slide 5-

S.I.R.S. Any Two of the Following Criteria Temperature: < 36.0, >38.0 Heart Rate : >90 Respiratory Rate: >20 WBC: <4,000, >12,000
Slide 6-

Sepsis Definition: SIRS plus evidence of local or systemic infection. Septic Shock Definition: Sepsis plus end organ hypoprofusion. Mortality of up to 40%
Slide 7-

Introduction Surgery, trauma, non-trauma local invasion can lead to bacterial insult. Once present, bacteria, initiate the host defense processes. Inflammatory mediators (kinins, histamine, etc.) are released, compliment and plasma proteins are released, PMN’s arrive, etc
Slide 8-

Risk Many established factors have a role in infection. These can be either surgical factors or patient-specific factors. Patient-specific factors can be further defined as either local or systemic
Slide 9-

Surgical Risk Factors Type of procedure Degree of contamination Duration of operation Urgency of operation
Slide 10-

SPREAD OF SURGICAL INFECTIONS NECROTIZING INFECTION ABSCESSES PHLEGMONS AND SURPERFICIAL INFECTIONS SPREAD OF INFECTIONS VIA THE LYMPHATIC SYSTEM SPREAD OF INFECTION VIA BLOODSREAM
Slide 11-

COMPLICATIONS OF SURGICAL INFECTION Fistulas and sinus tract Suppressed wound healing Immunosuppression and superinfection Bacteremia Organ dysfunctionSepsis, and systemic inflammatory response syndrome
Slide 12-

CLINICAL FINDIINGS AND DIAGNOSIS Physical examination Warmth, erythema, induration, tenderness Laboratory findings General findings: - leucocytosis, acidosis, and signs of disseminated intravascular coagulation - Cultures Imaging studies Source of infection
Slide 13-

TREATMENT Incision and drainage Excision Antibiotics Nutritional support
Slide 14-

Infections Two main types Community-Acquired Hospital-Acquired
Slide 15-

Community-Acquired Skin/soft tissue Cellulitis: Group A strep Carbubcles/furuncle: Staph aureus Necrotizing: Mixed Hiradenitis suppurativa: Staph aureus Lymphangitis: Staph aureus
Slide 16-

Cellulitis
Slide 17-

Cellulitis
Slide 18-

Cellulitis Definition: Diffuse infection with severe inflammation of dermal and subcutaneous layers of the skin Diagnosis: Pain, Warmth, Hyperesthesia Treatment: Antibiotics. Common Pathogens: Skin Flora (Streptococcus/Staphylococcus)
Slide 19-

FURUNCLES AND CARBUNCLES Furuncles and carbuncles are cutaneous abscess that begin in skin glands and hair follicles. If the pilosebaceous apparatus becomes obstructed at the skin level, the development of a furuncle can be anticipate A carbuncle is a deep –seated mass of fistulous tracts between infected hair follicles. Funruncles are the most common surgical infections, but carbuncles are rare
Slide 20-

Furuncle
Slide 21-

Carbuncle
Slide 22-

HIDRADENITIS Serious skin infection of the axillae or groin Consisting of multiple abscesses of the apocrine sweat glands. The condition often becomes chronic The cause is unknown but may involve a defect of terminal follicular epithelium
Slide 23-

Hiradenitis
Slide 24-

TREATMENT The classic therapy of furuncle is drainage, not antibiotics. Invasive carbuncles must be treated by excision and antibiotics. Hidradenitis is usually treated by drainage of the individual abscess and followed by careful hygeine
Slide 25-

Lymphangitis
Slide 26-

Community-Acquired Breast Abcess Staphylococcal infection Usually post-partum Treatment MRSA is uncommon
Slide 27-

Breast Abscess
Slide 28-

Abscess
Slide 29-

Abscess Definition: Infectious accumulation of purulent material (Neutrophils) in a closed cavity Diagnosis: Fluctuant: Moveable and compressible Treatment: Drainage
Slide 30-

Community-Acquired Peri-rectal abcess Results from infection of the anal crypts Can be extensive Can result in bacteremia Treatment
Slide 31-

Community-Acquired Hand Infections Paronychia Usually staph Where? Treatment Felon Where? Treatment Both can lead to tenosynovitis
Slide 32-

Paronychia An inflammatory reaction involving the folds of the skin surrounding the fingernail. It is characterized by acute or chronic purulent, tender, and painful swellings of the tissues around the nail, caused by an abscess of the nail fold. The pathogenic yeast causing paronychia is most frequently Candida albicans. The causative bacteria are usually Staphylococcus, Pseudomonas aeruginosa, or Streptococcus.
Slide 33-

Felon
Slide 34-

Community-Acquired
Slide 35-

DIFFUSE NECROTIZING INFECTIONS Particular dangerous Difficult to diagnose, extremely toxic, spread rapidly, often leading to limb amputation
Slide 36-

Pathogenic factors Anaerobic wound Bacterial exotoxins Bacterial synergy Thrombosis of nutrient bridging vessels
Slide 37-

Classification of diffuse necrotizing infections Clostridial Necrotizing cellulitis Myositis Nonclostridial Necrotizing fasciitis Streptococcal gangrene
Slide 38-

Clostridial Infections They are fastidious anaerobes On gram-stain they appear as relatively large, gram-positive, rod-shaped bacteria. A broad spectrum of disease is caused by clostridia
Slide 39-

Clinical Findings Crepitant abscess or cellulitis Invasion is usually superficial to the deep fascia and may spread very quickly, producing discoloration. Delayed debridement of injured tissue after devascularizing injury is the common setting.
Slide 40-

Gas Gangrene
Slide 41-

Clinical Findings Severe pain suggests extension into muscle compartments ( myositis). The disease progresses rapidly, with loss of blood supply to the infected tissue. Profound shock can appear early, rapidly leading to organ dysfunction. Air bubbles often visible on plain radiograph Crepitus may be present, but not reliable to differentiation .
Slide 42-

Nonclostridial Infections Caused by multiple nonclostridial bacterial pathogens. Microaerophilic streptococci, staphyloccci, aerobic gram-negative bacteria, and anaerobes, especially peptostreptococci and bacteroides.
Slide 43-

Necrotizing Soft Tissue Infection
Slide 44-

Necrotizing
Slide 45-

Clinical Findings Usually begins in a localized area such as a puncture wound, leg ulcer, or surgical wound. Externally, hemorrhgic bullae are usually the first sign of skin death The skin is anesthetic and crepitus is occasionally present. The fascial necrosis is usually wider than the skin appearance indicates. At operation, the finding of edematous, dull-gray, and necrotic fascia and subcutaneous tissue confirm the diagnosis.
Slide 46-

Streptococcal gangrene Group A streptococcus is a bacterium frequently found in in the skin and throat. Streptococcal gangrene is uncommon The sudden onset of severe pain is the most common presenting symptom, usually in an extremity associated with a wound. Fever and other signs of systemic infection are frequently present at the time of presentation. Shock and renal dysfunction are usually present within 24 hours.
Slide 47-

TREATMENT Complete debridement and depress tight fascial compartment. Amputation.
Slide 48-

TREATMENT Broad-spectrum antibiotic therapy Resuscitative therapy Treat diabetes mellitus aggressively Hyperbaric oxygenation inhibit bacterial invasion but does not eliminate the focus of infection.
Slide 49-

Community-Acquired Biliary Tract Usually result from obstruction Usual suspects: E. coli, Klebsiella, Enterococci Acute Cholecystitis GB empyema Ascending cholangitis Diagnosis Treatment
Slide 50-

Community-Acquired Peritonitis Causes Diagnosis Treatment
Slide 51-

Community-Acquired Viral Hepatitis HIV/AIDS
Slide 52-

Community-Acquired Tetanus C. tetani infection “ lock-jaw” Caused by exotoxin Treatment
Slide 53-

Post-Operative Infections Fever After Surgery The “Five W’s” Wind: Atelectisis Water: UTI Walking: DVT Wonder Drug: Medication Induced Wound: Surgical Site Infection
Slide 54-

Surgical Site Infections 3rd most common hospital infection Incisional Superficial Deep Organ Space Generalized (peritonitis) Abscess
Slide 55-

SSI – Definitions Infection Systemic and local signs of inflammation Bacterial counts ≥ 105 cfu/mL Purulent versus nonpurulent LOS effect Economic effect Surgical wound infection is SSI
Slide 56-

Superficial Incisional SSI Infection occurs within 30 days after the operation and involves only skin or subcutaneous tissue of the incision Mangram AJ et al. Infect Control Hosp Epidemiol. 1999;20:250-278.
Slide 57-

Deep Incisional SSI Infection occurs within 30 days after the operation if no implant is left in place or within 1 year if implant is in place and the infection appears to be related to the operation and the infection involves the deep soft tissue (e.g., fascia and muscle layers) Superficial incisional SSI Mangram AJ et al. Infect Control Hosp Epidemiol. 1999;20:250-278.
Slide 58-

Organ/Space SSI Infection occurs within 30 days after the operation if no implant is left in place or within 1 year if implant is in place and the infection appears to be related to the operation and the infection involves any part of the anatomy, other than the incision, which was opened or manipulated during the operation Deep incisional SSI Superficial incisional SSI Mangram AJ et al. Infect Control Hosp Epidemiol. 1999;20:250-278.
Slide 59-

SSI – Risk Factors Operation Factors Duration of surgical scrub Maintain body temp Skin antisepsis Preoperative shaving Duration of operation Antimicrobial prophylaxis Operating room ventilation Inadequate sterilization of instruments Mangram AJ et al. Infect Control Hosp Epidemiol. 1999;20:250-278. Foreign material at surgical site Surgical drains Surgical technique Poor hemostasis Failure to obliterate dead space Tissue trauma
Slide 60-

SSI – Risk Factors Patient Characteristics Age Diabetes HbA1C and SSI Glucose > 200 mg/dL postoperative period (<48 hours) Nicotine use: delays primary wound healing Steroid use: controversial Malnutrition: no epidemiological association Obesity: 20% over ideal body weight Mangram AJ et al. Infect Control Hosp Epidemiol. 1999;20:250-278. Prolonged preoperative stay: surrogate of the severity of illness and comorbid conditions Preoperative nares colonization with Staphylococcus aureus: significant association Perioperative transfusion: controversial Coexistent infections at a remote body site Altered immune response
Slide 61-

Preoperative preparation
Slide 62-

Perioperative Glucose Control Patients with a blood sugar > 300 mg/dL during or within 48 hours of surgery had more than 3X the likelihood of a wound infection
Slide 63-

Pre-operative Shaving
Slide 64-

Pre-operative shaving Shaving the surgical site with a razor induces small skin lacerations potential sites for infection disturbs hair follicles which are often colonized with S. aureus Risk greatest when done the night before Patient education be sure patients know that they should not do you a favor and shave before they come to the hospital!
Slide 65-

Influence of Shaving on SSI No Hair Group Removal Depilatory Shaved Number 155 153 246 Infection rate 0.6% 0.6% 5.6% Seropian. Am J Surg 1971; 121: 251
Slide 66-

Prophylactic Antibiotics Antibiotics given for the purpose of preventing infection when infection is not present but the risk of postoperative infection is present
Slide 67-

Prophylactic Antibiotics Questions Which cases benefit? Which drug should you use? When should you start? How much should you give? How long should antibiotics be continued?
Slide 68-

Surgical site prevention Use antibiotics appropriately Optimize oxygen tension Maintain normal Blood glucose Maintain normal Body temp Avoid shaving Site
Slide 69-

ppt slide no 69 content not found
Slide 70-

Treatment Incisional: open surgical wound, antibiotics for cellulitis or sepsis Deep/Organ space: Source control, antibiotics for sepsis
Slide 71-

Types of Surgery
Slide 72-

Operative Antibiotic Prophylaxis Decreases bacterial counts at surgical site Given within 30 minutes prior to starting surgery Vancomycin 1-2 hours prior to surgery Redose for longer surgery Do not continue beyond 24 hours
Slide 73-

ppt slide no 73 content not found
Slide 74-

Occupational Blood Bourne Virus Infections
Slide 75-

Q