Download Free Medical Respiratory Distress In Newborn PowerPoint Presentation

Respiratory Distress In Newborn PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical PowerPoint presentation titled "Respiratory distress in newborn” by Dr.mirzarahimi.

This medical PowerPoint presentation is about respiratory distress in newborns, a serious condition that requires urgent medical attention. It is characterized by difficulty breathing, rapid breathing, grunting sounds, flaring of the nostrils, chest retractions, and bluish discoloration of the skin. Respiratory distress can occur immediately after birth or develop in the first few hours or days of life.

The potential causes of respiratory distress in newborns include Transient tachypnea of the newborn (TTN), Meconium aspiration syndrome (MAS), Respiratory distress syndrome (RDS), Pneumonia, and Congenital heart disease.

Treatment for respiratory distress in newborns may include oxygen therapy, mechanical ventilation, medications to help with breathing, and treatment for any underlying conditions. It is important to seek medical attention immediately if you notice any signs of respiratory distress in your newborn, as early treatment can improve outcomes.

Tags : Respiratory distress in newborn, Transient tachypnea of the newborn, Meconium aspiration syndrome, Respiratory distress syndrome, Pneumonia, Congenital heart disease, Neonatal-Perinatal Medicine, Neonatal-Perinatal Medicine PowerPoint presentation

Published on : Jun 07, 2023 | Views : 1457 | Downloads : 2

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Respiratory distress in newborn Dr.mirzarahimi
Slide 2-

Neonatal Respiratory Distress Signs and symptoms Tachypnea (RR > 60/min) Nasal flaring Retraction Grunting Delayed or decreased air entry +/- Cyanosis +/- Desaturation
Slide 3-

ppt slide no 3 content not found
Slide 4-

Neonatal Respiratory Distress Etiologies Pulmonary causes RDS Pneumonia TTN MAS Other aspiration syndrome Air leak syndrome Lung hemorrhage Lung hypoplasia Congenital malformations Systemic causes Infections Metabolic causes Temperature Anemia Polycythemia Congenital heart disease Pulmonary hypertension Neuromuscular disorder Anatomic causes Upper airway obstruction Airway malformation Space occupying lesion Rib cage anomalies Phrenic nerve injury diagnosis : Hx, Phx and L.F
Slide 5-

Neonatal Respiratory DistressAlgorithm Respiratory Distress (tachypnoea, retractions, grunt) Preterm Term < 6hrs old > 6hrs old < 6hrs old > 6hrs old HMD (RDS) Pneumonia Lung anomaly Pneumonia CHD Pul. Hemorrhage TTN MAS/PPHN Asphyxia Lung anomaly Air leak Pneumonia CHD
Slide 6-

Respiratory Distress Syndrome
Slide 7-

Introduction The most frequent cause of respiratory distress in premature infants. 60-80% of <28wk GA ; 15-30% of 32-36wk GA ; 5% of 37wk-term. Classic presentation of grunting, retractions, increasing O2 requirement, reticulogranular pattern and air bronchograms on CXR and onset < 6hrs age
Slide 8-

Pathogenesis Prematurity Prenatal asphyxia Reduced surfactant synthesis, storage, release Increased alveolar surface tension Progressive atelectasis Diffusion Uneven V/Q Hypoventilation gradient Hypoxemia CO2 retention Acidosis Pulmonary vasoconstriction Hypoperfusion Capillary endothelial damage Plasma leak Fibrin
Slide 9-

Pathology Gross : Lung firm, red, liverlike Microscopic : Diffuse atelectasis, pink membrane lining alveoli & alveolar ducts. Pulmonary arterioles with thick muscular coat, small lumen. Distended lymphatics Electron microscopic : Damage / loss of alveolar epithelial cells, disappearance of lamellar inclusion bodies, swelling of capillary endothelial cells
Slide 10-

Pathology (contd.) Biophysical : Deficient / absent surfactant Abnormal pressure volume curve Normal Vol RDS Pressure Severely reduced arterial bed with blockage near pulmonary arterioles
Slide 11-

ppt slide no 11 content not found
Slide 12-

Pathology (contd.) Biochemical : Diminished surface-active phospholipid (phosphatidylcholine) Diminished apoprotein content ( SP-A, B, C, D)
Slide 13-

Pathophysiology Reduced lung compliance (1/5th -1/10th) Poor lung perfusion ( 50-60% not perfused), decreased capillary blood flow R--> L shunting ( 30-60% ) Alveolar ventilation decreased Lung volume reduced Increased work of breathing Hypoxemia, hypercapnia, acidosis
Slide 14-

Physiologic abnormalities Lung compliance 10-20% of norm Atelectasis…areas not ventilated Areas not perfused Decrease alveolar ventilation Reduce lung volume
Slide 15-

Risk factor Prematurity Acidosis Hypoxia Hypercapnia Hypothermia C/S Asphyxia and stress Male Familial DM mother
Slide 16-

signs tachypnea retraction grunting Nasal flaring apneic episode cyanosis extremities puffy or swollen
Slide 17-

Chest X-ray Ground glass appearance Reticulogranular With air bronchograms
Slide 18-

ppt slide no 18 content not found
Slide 19-

ppt slide no 19 content not found
Slide 20-

Treatment Surfactant Prevention rescue Supportive Thermal Fluid and nutrition oxygen Mechanical ventilation
Slide 21-

complications Pneumothorax PDA Infection Line problems ROP Chronic lung disease
Slide 22-

Meconium aspiration
Slide 23-

M .A .S آسپيريشن مايع آمنيوتيك آغشته به مكونيوم ممكن است منجر به سندرم آسپيريشن مكونيوم گردد كه مربيديتي و مورتاليتي قابل ملاحظه اي دارد بنابراين مديريت زايمان با مايع آمنيوتيك آغشته به مكونيوم براي پيشگيري از آسپيراسيون اهميت زيادي دارد
Slide 24-

تركيب مكونيوم Cellular particle Bile pigment Lango Mocus Vernix Pancreatic secretion One gr meconium = one mg Billirubin
Slide 25-

Incidence دفع مكونيوم 8 تا 20 درصد كل زايمانها ( متوسط 12 %) مكونيوم آسپيريشن در 4 درصد مكونيومي ها دیده می شود وجود دارد. SGA و Post maturityعمدتا
Slide 26-

فيزيوپاتولوژي اگر چه فيزيوپاتولوژي كامل آن و علت دفع مكونيوم كاملا شناخته نشده اما اين پديده بندرت قبل از هفته 34 ديده مي شود بسياري از مكونيوم دفع كرده ها علامتي دال بر مشكل تنفسي يا دپرسيون نداشته اند و عده اي هم بعلت آسفيكسي مكونيوم دفع كرده اند
Slide 27-

علت دفع مكونيوم 1) پديده فيزيولوژيك : تكامل عصبي پاراسمپاتيك و برقراري پريستالتيسم روده اي در پاسخ به تكامل جنين (شيوع در ترم ها و نادر بودن در نوزادان نارس ) 2) هيپوكسي : مي تواند باعث افزايش پريستالتيسم روده ها و كاهش تو ن اسفنكترآنال شود (البته اكثر نوزادان با مايع آمنيوتيك مكونيال آپگار پايين و اسيدوز ندارند )
Slide 28-

Alarm of MAS 1- Thick meconium 2-Fetal tachycardia 3- lack of increase heart rate during intra partum monitoring 4-Low cord PH
Slide 29-

پاتوژنز
Slide 30-

MAS complication Partial obstruction o complete obstruction : Surfactant destruction Chemical pneumonitis &Bacterial pneumonia Asphyxia PPHN
Slide 31-

Clinical sign Classic sign: Post maturity nail, skin , umblical cord are heavily stained with a yellowish pigment Early sign (resp . Distress) : grunting & cyanosis & nasal flaring & retraction & marked tachypnea Characteristic sign : chest overinflation and Rale
Slide 32-

Radiography of M.A.S Coarse , nodular , irregular pulmonary densities with areas of diminished aeration or consolidation. Hyperinflation of the chest . Atelectasis Flattening of diaphragm Cardiomegally (manifestation of the underlying prenatal hypoxia)
Slide 33-

Chest.X.Ray
Slide 34-

ppt slide no 34 content not found
Slide 35-

Meconium Aspiration Syndrome
Slide 36-

Meconium Aspiration Syndrome
Slide 37-

ABG in M.A.S شواهدي از يك آلكالوز تنفسي هيپوكسي در مواقع شديد اسيدوز تنفسي و اسيدوز متابوليك شواهدي از شنت راست به چپ
Slide 38-

Management of M.A.S حضور مكونيوم در مايع آمنيوتيك دليل بردیسترس جنینی نيست وچنانچه ضربان قلب جنین و پی-اچ بند ناف طبيعي باشد پيش آگهي خوب است آميخته شدن مكونيوم با مايع آمنيوتيك باضافه ضربان قلب نا مناسب نويد دهنده يك آسفيكسي مي باشد
Slide 39-

Intra partum در اين گونه مواقع وقتي سر بيرون آمد در روي پرينه بايد ساكشن دهان وبيني و فارنكس با كاتتر نمره 12 يا 14 انجام شود (قبل از اينكه توراكس بيرون بيايد و نوزاد بخواهد تنفس كند ) اولين ارزيابي نوزاد متولد شده : vigorous or depress
Slide 40-

Criteria of vigorous 1) Heart rate greater than 100 beat /min 2) Good muscle tone 3) regular breathing
Slide 41-

ppt slide no 41 content not found
Slide 42-

ppt slide no 42 content not found
Slide 43-

Guidelines of the baby exposed to meconium Vigorous Immediate tracheal suction Meconium No meconium reintubate and suction PPV and suction again later Clear secretions and meconium initial resuscitation steps HR>100 HR<100 No Yes The American Academy of Pediatrics Neonatal Resuscitation Program Steering Committee management guidelines of the baby exposed to meconium:
Slide 44-

ppt slide no 44 content not found
Slide 45-

ET suction indication Only in non vigorous baby - depressed respirations - decreased muscle tone - heart rate < 100 beats per minute Pharyngeal suctioning of an infant before delivery of the shoulders. Removal of meconium from hypopharynx and larynx by large-bore catheter. Meconium aspirator attached to wall suction. Endotracheal intubation for removal of meconium in the lower airway.
Slide 46-

Management Prevention Monitor fetal status Amnioinfusion Suctioning +/- intubation and immediate suctioning Avoid harmful techniques Intervention Optimal thermal environment & minimal handling Respiratory care, Oxygen therapy & ECMO Keep stable V/S Surfactant therapy
Slide 47-

Steroid therapy for meconium aspiration syndrome in newborn infants The Cochrane Database of Systematic Reviews 2007 Issue 3, The Cochrane Library (ISSN 1464-780X Conclusions: At present, there is insufficient evidence to assess the effects of steroid therapy in the management of meconium aspiration syndrome (no significant reduction in mortality, duration of hospital stay, Duration of mechanical ventilation, incidence of air leak,increase in duration of oxygen therapy was seen with the use of steroids)
Slide 48-

Role of antibiotics in meconium aspiration syndrome Ann Trop Paediatr. 2007 Jun;27(2):107-13. Basu S, Kumar A, Bhatia BD. Division of Neonatology, Department of Paediatrics, Institute of Medical Sciences, Banaras Hindu University, Varanasi, India. drsriparnabasu@rediffmail.com CONCLUSION: Routine antibiotic therapy is not necessary for managing MAS. No significant difference period of oxygen dependency (5.8 vs 5.9 days) day of starting feeds (4.0 vs 4.2) day of achievement of full feeds (9.4 vs 9.3) clearance of chest radiograph (11.7 vs 12.9 days) duration of hospital stay (13.7 vs 13.5 days)
Slide 49-

Surfactant for meconium aspiration syndrome in full term/near term infants Cochrane Database Syst Rev. 2007 Jul 18;(3):CD002054 El Shahed A, Dargaville P, Ohlsson A, Soll R. CONCLUSIONS: In infants with MAS, surfactant administration may reduce the severity of respiratory illness and decrease the number of infants with progressive respiratory failure requiring support with ECMO. The relative efficacy of surfactant therapy compared to, or in conjunction with, other approaches to treatment including inhaled nitric oxide, liquid ventilation, surfactant lavage and high frequency ventilation remains to be tested.
Slide 50-

PPHN prevention Avoid vasoconstriction Acidosis Hypoxia Metabolic disturbance - Hypocalcemia - Hypercalcemia - Hyperglycemia - Hypoglycemia Prevent right to left shunt
Slide 51-

Infections
Slide 52-

Infections Pneumonia & Sepsis have various manifestations including typical signs of distress as well as temperature instability. Common pathogen- Group B Streptococcus, Staph aureus, Streptococcus Pneumonia,Gm neg. rods
Slide 53-

Infections con.. Risk factors- prolonged rupture of membranes, prematurity,& maternal fever. CXR- bilateral infiltrates suggesting in utero infection.
Slide 54-

Congenital pneumonia Sepsis risk factors PROM Prematurity Maternal fever, discharge, abdominal pain, leukocytosis Colonization with GBS Same signs of RDS X-ray
Slide 55-

ppt slide no 55 content not found
Slide 56-

Transient Tachypnea of Newborn Most common cause of respiratory distress. Residual fluid in fetal lung tissues. Risk factors- maternal asthma, c- section, male sex, macrosomia, maternal diabetes
Slide 57-

TTN Tachypnea immediately after birth or within two hours, with other predictable signs of respiratory distress. Symptoms can last few hours to two days. Chest radiography shows diffuse parenchymal infiltrates, a “ wet silhouette” around heart, or intralobar fluid accumulation
Slide 58-

X-ray Fluid in the fissure
Slide 59-

Transient tachypnea of newborn Term Cesarian delivery Usually tachypnea without O2 requirment Resolve in 48-72 houres Lung fluid X-ray
Slide 60-

Other causes- Congenital malformations-Pulmonary hypoplasia, congenital emphysema, esophageal atresia & diaphragmatic hernia. Neurological causes- hydrocephalus & intracranial hemorrhage. Metabolic derangements-hypoglycemia, hypocalcemia, polycythemia.
Slide 61-

Congenital Heart disease Cyanotic Heart Disease- Tetralogy of fallot- ( VSD, Pulmonary stenosis, overriding aorta, RVH) Tricuspid atresia Transposition of great vessel Total anomalous pul. venous return Truncus arteriosus.
Slide 62-

Hyperoxia Test Obtain ABG–> Then place the patient on 100% O2 for 10 minutes then repeat ABG , If the cyanosis is pulmonary , the PaO2 should be increased by 30 mm of Hg. If the cause is cardiac , there will be minimal improvement in PaO2.