Download Free Medical Anal Canal Fissure In ANO Haemorrhoids PowerPoint Presentation

Anal Canal Fissure In ANO Haemorrhoids PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical PowerPoint presentation titled "Anal Canal Fissure In Ano Haemorrhoids” by Dr.Amit Gupta.

This medical PowerPoint presentation talks about anal canal fissure and haemorrhoids, two distinct conditions that can affect the anal region.

Anal Canal Fissure: An anal fissure is a small tear or cut in the lining of the anal canal. It is usually caused by passing hard or large stools, chronic constipation, or diarrhea. The main symptom of an anal fissure is pain, which is usually severe and can be accompanied by bleeding. Other symptoms may include itching, discomfort, and a burning sensation. Anal fissures can usually be treated with stool softeners, sitz baths, and topical medications. In severe cases, surgery may be necessary.

Haemorrhoids: Hemorrhoids, also known as piles, are swollen veins in the anus or lower rectum. They can be internal or external and may cause pain, itching, bleeding, and discomfort during bowel movements. Hemorrhoids can be caused by straining during bowel movements, pregnancy, obesity, or a sedentary lifestyle. Treatment options for hemorrhoids include over-the-counter creams and ointments, sitz baths, increased fiber intake, and surgery in severe cases.

While anal fissures and hemorrhoids can both cause pain and discomfort in the anal region, they are distinct conditions with different causes and treatments. If you are experiencing symptoms in this area, its important to speak with your healthcare provider to determine the underlying cause and appropriate treatment.

Tags : anal canal fissure, haemorrhoids, Gastroenterology, Gastroenterology PowerPoint presentation

Published on : May 02, 2023 | Views : 1904 | Downloads : 5

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Anal Canal Fissure In AnoHaemorrhoids Dr.Amit Gupta Associate Professor Dept. of Surgery
Slide 2-

Anorectal Anatomy Anal verge Anal canal Arterial Supply Inferior rectal A middle rectal A Venous drainage Inferior rectal V middle rectal V 3 hemorrhoidal complexes L lateral R antero-lateral R posterolateral Lymphatic drainage Above dentate: Inf. Mesenteric Below dentate: internal iliac Nerve Supply Sympathetic: Superior hypogastric plexus Parasympathetic: S234 (nerviergentis Pudendal Nerve: Motor and sensory
Slide 3-

External Internal Anoderm Swell, discomfort, difficult hygiene Pain? -> Thrombosed Pain? -> painless Bright red bleeding Prolapse associated with defecation
Slide 4-

Background They are part of the normal anoderm cushions They are areas of vascular anastamosis in a supporting stroma of subepithelial smooth muscles. The contribute 15-20% of the normal resting pressure and feed vital sensory information . 3 main cushions are found L lateral R anterior R posterior But can be found anywhere in anus Prevalence is 4% Miss labelling by referring physicians and patients is common This combination is only in 19%
Slide 5-

3 main processes: Increased venous pressure Weakness in supporting fibromuscular stroma Increased internal sphincter tone Pathogenesis
Slide 6-

Risk factors
Slide 7-

Classification
Slide 8-

Thrombosed external piles
Slide 9-

First-degree internal piles viewed through anoscope
Slide 10-

Second-degree internal prolapsed piles, reduced spontaneously
Slide 11-

Third-degree internal prolapsed piles, requiring manual reduction
Slide 12-

Fourth-degree strangulated internal and thrombosed external piles
Slide 13-

Clinical assessment
Slide 14-

Lab: CBC / Clotting profile/ Group and save Proctography: if rectal prolpse is suspected Colonoscopy: if higher colonic or sinister pathology is suspected Investigations
Slide 15-

Thrombosed internal haemorrhoids Thrombosed external haemorrhoids Complications Ulceration Thrombosis Sepsis and abscess formation Incontinence
Slide 16-

ppt slide no 16 content not found
Slide 17-

ppt slide no 17 content not found
Slide 18-

Internal Haemorrhoids Treatment
Slide 19-

ppt slide no 19 content not found
Slide 20-

Excision of thrombosed external hemorrhoid.
Slide 21-

Closed hemorrhoidectomy
Slide 22-

Grade 4 hemorrhoid before reduction Placement of stapling device obturator Stapling device
Slide 23-

If presentation less than 72 hours: Enucleate under LA or GA Leave wound open to close by secondary intension Apply pressure dressing for 24 hours post op If more than 72 hours: Conservative measures External Haemorrhoids Treatment
Slide 24-

Anal Fissure Linear tears in the anal mucosa exposing the internal sphincter 90% are posterior
Slide 25-

Young & middle aged adults Male = Female Location – posterior midline (most common) Anterior midline fissures – more common in females In any event, length of each fissure is remarkably constant, extending from the dentate line to the anal verge and corresponding roughly to the lower half of the internal sphincter
Slide 26-

Pathology Acute fissures – heal promptly with conservative treatment Secondary changes if present, it does not heal readily Sentinel pile Hypertrophied anal papilla Long standing Fibrous induration in lateral edges of fissure Fibrosis at the base of ulcer (internal sphincter) At any stage Frank suppuration – intersphincteric / perianal abscess
Slide 27-

Etiology Initiation – trauma Why midline posterior fissures are more common? Dietary factors Decreased risk – raw foods, vegetables, whole grain bread Increased risk – white bread sausages etc. Secondary fissure Crohn’s disease Previous anal surgery, especially hemorrhoidectomy Fistula-in-ano surgery Anterior fissure in females resulting from childbirth Long standing loose stools with chronic laxative abuse
Slide 28-

Initiation – trauma Perpetuation of fissure – abnormality of internal anal sphincter Higher resting pressure within the internal anal sphincter in pts with fissures than in normal control Rectal distension  reflex relaxation of internal anal sphincter  overshoot contractions in these patients  sphincter spasm and pain Elevated sphincter pressures cause ischemia of the anal lining resulting in pain and failure to heal Posterior commissure perfused more poorly than the other portion of the anal canal
Slide 29-

Clinical Features Pain and spasm Sharp, agonizing during defecation, recurrent, worsens constipation. Bleeding In small amounts, approximately 70% of patients note bright red blood on the toilet paper or stool Discharge Irritation and pruritis ani due to malodorous discharge of the pus Constipation Painless non-healing fissure with occasional bleed – may be a progenitor of IBD
Slide 30-

Diagnosis Inspection – Acute fissure is seen as Linear tear most important Palpation Anoscopy Sigmoidoscopy Biopsy
Slide 31-

Differential diagnosis Anorectal suppuration Pruritus ani Fissure in inflammatory bowel disease Carcinoma Syphilitic fissures Tuberculous ulcer Anal abrasion
Slide 32-

Treatment Acute Fissure Stool-bulking agents Warm sitz bath Healed (80%) Non-healed (20%) 0.2% Nitroglycerin or 2% Nifedipine or Diltiazem 4-6 wks Repeat treatment with alternate medication Non-healed (30%) Healed (70%) Chronic fissure Botulinum toxin A injection Open LIS 98% healing Closed LIS
Slide 33-

Acute anal fissure: Spontaneous healing, High fiber diet, adequate water intake and warm sitz bath, stool softener/bulk laxative, suppositories Sodium tetradecyl sulphate
Slide 34-

Chronic anal fissure: Conservative Surgical
Slide 35-

Pharmacological Sphincterotomy Pharmacological manipulation of anal sphincter tone as an alternative modality to surgery for the treatment of anal fissure Shares the same goal as lateral sphincterotomy without its possible long-term side effects Pharmacological agents lower anal canal resting pressure producing chemical sphincterotomy without causing permanent damage to the anal sphincter mechanism
Slide 36-

By enhancing internal anal sphincter (IAS) relaxation via nitric oxide donation intracellular Ca2+ depletion muscarinic receptor stimulation adrenergic inhibition This improves blood supply at the site of the fissure that would promote healing of anal fissures Nitric oxide donors and calcium channel blockers, agents that directly reduce resting anal pressure, has now largely replaced traditional surgical methods as first-line treatment for chronic anal fissure
Slide 37-

Botulinum Toxin A L-arginine Gonyautoxin Topical sildenafil Other Agents